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Relation Between HbF,Hematological Parameters & XmnI Gene Polymorphism farm hand forged spring steel sickle king chai sickle weeding knife grinding the blade free firewood
Sickle cell disease is a major public health concern in the state of Chhattisgarh, India. Sickle cell disease is a recessive inherited structural disorder of hemoglobin. The abnormal hemoglobin causes distorted shapes (which appear sickle like) red blood cells. These abnormal RBCs are fragile and prone to rupture. Normal hemoglobin is called Hb A, but people with sickle cell disease have only Hb S. When fetal hemoglobin production is turned off after birth, normal people begin to produce adult hemoglobin (HbA). Children with sickle-cell disease instead begin producing a defective form of hemoglobin called hemoglobin S.If fetal hemoglobin remains the predominant form of hemoglobin after birth, the number of painful episodes decreases in patients with sickle-cell disease. Xmn I polymorphism is a factor that has been found to increase fetal hemoglobin production.
4839 RUR
Painful crises in sickle cell anemia farm hand forged spring steel sickle king chai sickle weeding knife grinding the blade free firewood
Sickle cell anemia is one of the major hereditary health problem in tropical Africa where the sickle cell gene frequency is particularly high. The disease occurs with high incidence in west,east and central Africa and this has been related to the prevalence of malaria in these regions. The classic hallmark of sickle cell anemia are episodes of vaso-occlusive crisis often with multiorgan manifestation and chronic haemolytic anemia. vaso-occlusive crisis is the most common cause of hospitalization in sickle cell disease. It affects the growth and academic performance of individual. Thus it would be quite helpful to terminate such painful episodes at a very early stage. Serum CPK,LDH and 24 hour urinary creatinine can be used as potential markers of tissue ischemia in such painful episodes.
5490 RUR